1: Westphal G, Hallier E.
Mercury in infants given vaccines containing thiomersal.
Lancet. 2003 Feb 22;361(9358):699; author reply 699. No abstract available.
2: Arch Toxicol 2003 Jan;77(1):50-5
Thimerosal induces micronuclei in the cytochalasin B block micronucleus test
with human lymphocytes.
Westphal GA, Asgari S, Schulz TG, Bunger J, Muller M, Hallier E.
Department of Occupational Health, Georg-August-University Gottingen, Waldweg
37, 37073 Gottingen, Germany, gwestph@gwdg.de
Thimerosal is a widely used preservative in health care products, especially in
vaccines. Due to possible adverse health effects, investigations on its
metabolism and toxicity are urgently needed. An in vivo study on chronic
toxicity of thimerosal in rats was inconclusive and reports on genotoxic effects
in various in vitro systems were contradictory. Therefore, we reinvestigated
thimerosal in the cytochalasin B block micronucleus test. Glutathione
S-transferases were proposed to be involved in the detoxification of thimerosal
or its decomposition products. Since the outcome of genotoxicity studies can be
dependent on the metabolic competence of the cells used, we were additionally
interested whether polymorphisms of glutathione S-transferases (GSTM1, GSTT1, or
GSTP1) may influence the results of the micronucleus test with primary human
lymphocytes. Blood samples of six healthy donors of different glutathione
S-transferase genotypes were included in the study. At least two independent
experiments were performed for each blood donor. Significant induction of
micronuclei was seen at concentrations between 0.05-0.5 micro g/ml in 14 out of
16 experiments. Thus, genotoxic effects were seen even at concentrations which
can occur at the injection site. Toxicity and toxicity-related elevation of
micronuclei was seen at and above 0.6 micro g/ml thimerosal. Marked individual
and intraindividual variations in the in vitro response to thimerosal among the
different blood donors occurred. However, there was no association observed with
any of the glutathione S-transferase polymorphism investigated. In conclusion,
thimerosal is genotoxic in the cytochalasin B block micronucleus test with human
lymphocytes. These data raise some concern on the widespread use of thimerosal.
3: Int J Hyg Environ Health 2001 Jul;203(5-6):479-81
Inhibition of the human erythrocytic glutathione-S-transferase T1 (GST T1) by
thimerosal.
Muller M, Westphal G, Vesper A, Bunger J, Hallier E.
Department of Occupational and Social Medicine, Georg-August-University
Gottingen, D-37073 Gottingen, Germany.
mmuelle3@gwdg.de
We have investigated the interaction of thimerosal, a widely used antiseptic and
preservative, with the human erythrocytic GST T1 (glutathione-S-transferase T1).
This detoxifying enzyme is expressed in the erythrocytes of solely the human
species and it displays a genetic polymorphism. Due to this polymorphism about
25% of the individuals of the caucasian population lack this activity
("non-conjugators"), while 75% show it ("conjugators") (Hallier, E., et al.,
1993). Using our newly developed HPLC-fluorescence detection assay (Muller, M.,
et al., 2001) we have profiled the kinetics of enzyme inhibition in erythrocyte
lysates of two individuals previously identified as "normal conjugator" (medium
enzyme activity) and "super-conjugator" (very high activity). For the normal
conjugator we have determined a 2.77 mM thimerosal concentration to inhibit 50%
of the GST T1 activity. In the case of the super-conjugator a 2.3 mM thimerosal
concentration causes a 50% inhibition of the enzyme activity. For both
phenotypes a 14.8 mM thimerosal concentration results in residual enzyme
activities equal to those typically detected in non-conjugator lysates. Thus,
sufficiently high doses of thimerosal may be able to change the phenotypic
status of an individual--at least in vitro--by inhibition of the GST T1 enzyme.
4: Int Arch Occup Environ Health 2000 Aug;73(6):384-8
Homozygous gene deletions of the glutathione S-transferases M1 and T1 are
associated with thimerosal sensitization.
Westphal GA, Schnuch A, Schulz TG, Reich K, Aberer W, Brasch J, Koch P,
Wessbecher R, Szliska C, Bauer A, Hallier E.
Abteilung fur Arbeits- und Sozialmedizin, Georg-August-Universitat Gottingen,
Germany. gwestph@gwdg.de
OBJECTIVE: Thimerosal is an important preservative in vaccines and
ophthalmologic preparations. The substance is known to be a type IV sensitizing
agent. High sensitization rates were observed in contact-allergic patients and
in health care workers who had been exposed to thimerosal-preserved vaccines.
There is evidence for the involvement of the glutathione system in the
metabolism of thimerosal or its decomposition products (organomercury alkyl
compounds). Thus detoxification by polymorphically expressed glutathione
S-transferases such as GSTT1 and GSTM1 might have a protective effect against
sensitization by these substances. METHODS: To address this question, a case
control study was conducted, including 91 Central European individuals with a
positive patch-test reaction to thimerosal. This population was compared with
169 healthy controls and additionally with 114 individuals affected by an
allergy against para-substituted aryl compounds. The latter population was
included in order to test whether possible associations were due to
substance-specific effects, or were a general feature connected with type IV
immunological diseases. Homozygous deletions of GSTT1 and GSTM1 were determined
by polymerase chain reaction. RESULTS: Glutathione S-transferase M1 deficiency
was significantly more frequent among patients sensitized to thimerosal (65.9%,
P = 0.013) compared with the healthy control group (49.1%) and the
"para-compound" group (48%, P = 0.034). Glutathione S-transferase T1 deficiency
in the thimerosal/mercury group (19.8%) was barely elevated versus healthy
controls (16.0%) and the "para-compound" group (14.0%). The combined deletion
(GSTT1-/GSTM1-) was markedly more frequent among thimerosal-sensitized patients
than in healthy controls (17.6% vs. 6.5%, P = 0.0093) and in the "para-compound"
group (17.6% vs. 6.1%, P =0.014), revealing a synergistic effect of these enzyme
deficiencies (healthy controls vs. thimerosal GSTM1 negative individuals, OR =
2.0 [CI = 1.2-3.4], GSTT1-, OR = 1.2 [CI = 0.70-2.1], GSTM1/T1-, OR = 3.1 [CI =
1.4-6.5]). CONCLUSIONS: Since the glutathione-dependent system was repeatedly
shown to be involved in the metabolism of thimerosal decomposition products, the
observed association may be of functional relevance.
5: Lancet 2002 Nov 30;360(9347):1737-41
Comment in:
Lancet. 2002 Nov 30;360(9347):1711-2.
Lancet. 2003 Feb 22;361(9358):698-9; author reply 699.
Lancet. 2003 Feb 22;361(9358):698; author reply 699.
Lancet. 2003 Feb 22;361(9358):699; author reply 699.
Mercury concentrations and metabolism in infants receiving vaccines containing
thiomersal: a descriptive study.
Pichichero ME, Cernichiari E, Lopreiato J, Treanor J.
Department of Microbiology/Immunology, University of Rochester, Rochester, New
York, NY, USA.
michael_pichichero@urmc.rochester.edu
BACKGROUND: Thiomersal is a preservative containing small amounts of
ethylmercury that is used in routine vaccines for infants and children. The
effect of vaccines containing thiomersal on concentrations of mercury in
infants' blood has not been extensively assessed, and the metabolism of
ethylmercury in infants is unknown. We aimed to measure concentrations of
mercury in blood, urine, and stools of infants who received such vaccines.
METHODS: 40 full-term infants aged 6 months and younger were given vaccines that
contained thiomersal (diptheria-tetanus-acellular pertussis vaccine, hepatitis B
vaccine, and in some children Haemophilus influenzae type b vaccine). 21 control
infants received thiomersal-free vaccines. We obtained samples of blood, urine,
and stools 3-28 days after vaccination. Total mercury (organic and inorganic) in
the samples was measured by cold vapour atomic absorption. FINDINGS: Mean
mercury doses in infants exposed to thiomersal were 45.6 microg (range
37.5-62.5) for 2-month-olds and 111.3 microg (range 87.5-175.0) for
6-month-olds. Blood mercury in thiomersal-exposed 2-month-olds ranged from less
than 3.75 to 20.55 nmol/L (parts per billion); in 6-month-olds all values were
lower than 7.50 nmol/L. Only one of 15 blood samples from controls contained
quantifiable mercury. Concentrations of mercury were low in urine after
vaccination but were high in stools of thiomersal-exposed 2-month-olds (mean 82
ng/g dry weight) and in 6-month-olds (mean 58 ng/g dry weight). Estimated blood
half-life of ethylmercury was 7 days (95% CI 4-10 days). INTERPRETATION:
Administration of vaccines containing thiomersal does not seem to raise blood
concentrations of mercury above safe values in infants. Ethylmercury seems to be
eliminated from blood rapidly via the stools after parenteral administration of
thiomersal in vaccines.
http://www.altcorp.com/TESTFoundation/thimstudys.htm
A. Toxic
Effects of Thimerosal on Vital Mammalian Enzymes and Enzyme Systems
- Rat brain (Na+-K+)ATPase: modulation of its oubain-sensitive K+-PNPPase
activity by thimerosal. Lewis and Bowler (1983). Int. J. Biochem. 15:5-7.
(6298022)
- Modified cation activation of the (Na+K+)-ATPase following
treatment with thimerosal. Kaplan and Mone (1985). Arch. Biochem. Biophys. 237:386-395.
(2983612)
- The effects of organic and inorganic mercuric salts on (Na+K+)ATPase
in different cerebral fractions in control and intrauterine growth-retarded rats:
alterations induced by serotonin. Chanez et al., (1989). Neurotoxicology 10:699-706.
(2562765)
- Kinetics of merthiolate-induced aggregation of human platelets. Vrzheshch et al.,
(1992). Thromb. Res. 67:505-516. (1448785)
- The effects of thimerosal on calcium uptake and inositol 1,4,5-triphosphate-induced
calcium release in cerebellar microsomes. Sayers et al., (1993). Biochem. J. 289:883-887.
(8435083)
- Thimerosal interacts with the Ca2+ release channel ryanodine receptor from
skeletal muscle sarcoplasmic reticulum. Abramson et al., (1995). J. Biol. Chem. 270:29644-29647.
(8530347)
- Effect of thimerosal and other sulfhydryl reagents on calcium permeability in thymus
lymphocytes. Pintado et al., (1995). Biochem. Pharmacol. 49:227-232.
(7840800)
- Increased expression of procoagulant activity on the surface of human platelets exposed
to heavy metal compounds. Goodwin et al., (1995). Biochem. J. 308:15-21.
(7755558)
- Effects of thimerosal, an organic sulfhydryl modifying agent, on serotonin transport
activity into rabbit blood platelets. Nishio et al., (1996). Neurochem. Int. 29:391-394.
(8939447)
- Effects of thimerosal on the transient kinetics of inositol 1,4,5-triphosphate-induced
Ca2+ release from cerebellar microsomes. Mezna and Michelangeli (1997). Biochem.
J. 325:177-182. (9224644)
- Effects of thiol-modifying agents on a K(Ca2+) channel of intermediate
conductance in bovine aortic endothelial cells. Cai and Sauve (1997). J. Membr. Biol.
158:147-158. (9230092)
- Thimerosal: a versatile sulfhydryl reagent, calcium mobilizer, and cell
function-modulating agent. Elferink (1999). Gen. Pharmacol. 33:1-6.
(10428009)
- Cardiac ryanodine receptor activity is altered by oxidizing reagents in either the
luminal or cytoplasmic solution. Eager and Dulhunty (1999). J. Membr. Biol. 167:205-214.
(9929372)
- Modulation of type 1, 2 and 3 inositol 1,4,5-triphosphate receptors by cyclic ADP-ribose
and thimerosal. Vanlingen et al., (1999). Cell Calcium 25:107-114.
(10326677)
- Thimerosal enhances agonist-specific differences between [Ca2+]i
oscillations induced by phenylephrine and ATP in single rat hepatocytes. Green et al.,
(1999). Cell Calcium 25:173-178. (10326684)
B. Chromosomal
Aberrations Induced by Thimerosal Exposure
- Analysis of nine known or suspected spindle poisons for mitotic chromosome
malsegregation using Saccharomyces cerevisiae D61.M. Albertini S (1990).
Mutagenesis 6:65-70. (2263203)
- Effects of 10 known or suspected spindle poisons in the in vitro porcine brain tubulin
assembly assay. Brunner et al., (1991). Mutagenesis 6:65-70. (2038274)
- In vitro studies with nine known or suspected spindle poisons: results in tests for
chromosome malsegregation in Aspergillus nidulans.Crebelli et al., (1991). Mutagenesis
6:131-136. (2056914)
- The detection and assessment of the aneugenic potential of environmental chemicals: the
European Community Aneuploidy Project. Parry and Sors. (1993).Mutat Res 287:3-15.
(7683383)
- Effects of potential anueploidy inducing agents on microtubule assembly in vitro. Wallin
and Hartley-Asp (1993). Mutat. Res. 287:17-22. (7683380)
- An evaluation of the use of in vitro tubulin polymerization, fungal and wheat assays to
detect the activity of potential chemical aneugens. Parry (1993). Mutat. Res. 287:23-28.
(7683381)
- A comparison of two in vitro mammalian cell cytogenetic assays for the detection of
mitotic aneuploidy using 10 known or suspected aneugens. Warr et al., (1993). Mutat.
Res. 287:29-46. (7683382)
- Induction of mitotic aneuploidy using Chinese hamster primary embryonic cells. Test
results of 10 chemicals. Natarajan et al., (1993). Mutat. Res. 287:47-56.
(7683384)
- C-mitosis and numerical chromosome aberration analyses in human lymphocytes: 10 known or
suspected spindle poisons. Sbrana et al., (1993). Mutat. Res. 287:57-70.
(7683385)
- The cytochalasin-B micronucleus/kinetochore assay in vitro.: studies with 10 suspected
aneugens. Lynch and Parry (1993). Mutat. Res. 287:71-86. (7683386)
- An overview of the results of testing of known or suspected aneugens using mammalian
cells in vivo. Natarajan (1993).Mutat. Res. 287:113-118. (7683377)
- In vivo studies on chemically induced anueploidy in mouse somatic and germinal cells.
Leopardi et al., (1993). Mutat. Res. 287:119-130. (7683378)
- Synopsis of the in vivo results obtained with the 10 known or suspected aneugens tested
in the CEC collaborative study. Adler (1993). Mutat. Res. 287:131-137.
(7683379)
- Micronucleus test and metaphase analyses in mice exposed to known and suspected spindle
poisons. Marrazzini et al., (1994). Mutagenesis 9:505-515. (7854141)
C. Disruption of Calcium Homeostasis by Thimerosal
- Modulation of inositol 1,4,5-trisphosphate binding to the various inositol
1,4,5-trisphosphate receptor isoforms by thimerosal and cyclic ADP-ribose. Vanlingen et
al., (2001) Biochemical Pharmacology 61:803-809. (11274965)
- Thimerosal: a versatile sulfhydryl reagent, calcium mobilizer, and cell
function-modulating agent. Elferink JG (1999). Gen Pharmacol 33:1-6.
(10428009)
- Thimerosal enhances agonist-specific differences between [Ca2+]i oscillations
induced by phenylephrine and ATP in single rat hepatocytes. Green AK, Cobbold PH, Dixon CJ
(1999). Cell Calcium 25:173-8. (10326684)
- Modulation of type 1, 2 and 3 inositol 1,4,5-trisphosphate receptors by cyclic
ADP-ribose and thimerosal. Vanlingen S, Sipma H, Missiaen L, De Smedt H, De Smet P,
Casteels R, Parys JB (1999). Cell Calcium 25:107-14. (10326677)
- Effects of thimerosal on the transient kinetics of inositol 1,4,5-trisphosphate-induced
Ca2+ release from cerebellar microsomes. Mezna M, Michelangeli F (1997).
Biochem J 325:177-82. (9224644)
- Effects of thimerosal, an organic sulfhydryl modifying agent, on serotonin transport
activity into rabbit blood platelets. Nishio H, Nezasa K, Hirano J, Nakata Y (1996).
Neurochem Int 29:391-6. (8939447)
- Thimerosal-induced Ca2+ mobilization in isolated guinea pig cochlear outer
hair cells. Chen L, Harada N, Yamashita T (1998). Acta Otolaryngol Suppl 539:28-33.
(10095857)
- Phosphatidylserine synthesis in glioma C6 cells is inhibited by Ca2+
depletion from the endoplasmic reticulum: effects of 2,5-di-tert-butylhydroquinone and
thimerosal. Wiktorek M, Sabala P, Czarny M, Baranska J (1996). Biochem Biophys Res
Commun 224:645-50. (8713102)
- Thimerosal interacts with the Ca2+ release channel ryanodine receptor from
skeletal muscle sarcoplasmic reticulum. Abramson JJ, Zable AC, Favero TG, Salama G (1995).
J Biol Chem 270:29644-7. (8530347)
- Thimerosal increases the responsiveness of the calcium receptor in human parathyroid and
rMTC6-23 cells. Mihai R, Lai T, Schofield G, Farndon JR (1999). Cell Calcium
26:95-101. (10598273)
- Effect of thimerosal and other sulfhydryl reagents on calcium permeability in thymus
lymphocytes. Pintado E, Baquero-Leonis D, Conde M, Sobrino F (1995). Biochem Pharmacol
49:227-32. (7840800)
- Effect of thimerosal on cytosolic calcium and phosphatidylserine synthesis in Jurkat T
cells. Pelassy C, Breittmayer JP, Ticchioni M, Aussel C (1994). Int J Biochem
26:93-6. (8138053)
- Reversible blockade of the calcium-activated nonselective cation channel in brown fat
cells by the sulfhydryl reagents mercury and thimerosal. Koivisto A, Siemen D, Nedergaard
J (1993). Pflugers Arch 425:549-51. (7510880)
- Thimerosal induced changes of intracellular calcium in human endothelial cells. Gericke
M, Droogmans G, Nilius B (1993). Cell Calcium 14:201-7. (8500136)
- The effects of thimerosal on calcium uptake and inositol 1,4,5-trisphosphate-induced
calcium release in cerebellar microsomes. Sayers LG, Brown GR, Michell RH, Michelangeli F
(1993). Biochem J 289:883-7 (8435083)
- The effects of thimerosal and cyclopiazonic acid on the Ca(2+)-pumps from rat
cerebellum microsomes. Michelangeli F, DaSilva A, Sayers L, Brown G (1992). Biochem
Soc Trans 20:205S. (1327911)
- The thiol reagent, thimerosal, evokes Ca2+ spikes in HeLa cells by
sensitizing the inositol 1,4,5-trisphosphate receptor. Bootman MD, Taylor CW, Berridge MJ
(1992). J Biol Chem 267:25113-9. (1334081)
- Cytosolic Ca2+ spikes evoked by the thiol reagent thimerosal in both intact
and internally perfused single pancreatic acinar cells. Thorn P, Brady P, Llopis J,
Gallacher DV, Petersen OH (1992). Pflugers Arch 422:173-8.
(1336850)
- Effect of the sulfhydryl reagent thimerosal on cytosolic free Ca2+ and
membrane potential of thymocytes. Gukovskaya AS, Trepakova ES, Zinchenko VP, Korystov YN,
Bezuglov VV (1992). Biochim Biophys Acta 1111:65-74. (1390866)
- Thimerosal blocks stimulated but not basal release of endothelium-derived relaxing
factor (EDRF) in dog isolated coronary artery. Crack P, Cocks T (1992). Br J Pharmacol
107:566-72. (1384915)
- Thimerosal induces calcium mobilization, fructose 2,6-bisphosphate synthesis and
cytoplasmic alkalinization in rat thymus lymphocytes. Martin F, Gualberto A, Sobrino F,
Pintado E (1991). Biochim Biophys Acta 1091:110-4. (1995061)
- Involvement of calcium in the thimerosal-stimulated formation of leukotriene by fMLP in
human polymorphonuclear leukocytes. Hatzelmann A, Haurand M, Ullrich V (1990). Biochem
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- The effect of thimerosal on neutrophil migration: a comparison with the effect on
calcium mobilization and CD11b expression. Elferink JG, de Koster BM (1998). Biochem
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- Enhancement of eicosanoid synthesis in mouse peritoneal macrophages by the organic
mercury compound thimerosal. Kaever V, Goppelt-Strube M, Resch K (1988).
Prostaglandins 35:885-902. (3141973)
- The sulfhydryl reagent thimerosal elicits human platelet aggregation by mobilization of
intracellular calcium and secondary prostaglandin endoperoxide formation. Hecker M, Brune
B, Decker K, Ullrich V (1989). Biochem Biophys Res Commun 159:961-8
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- Platelet aggregation by thimerosal: role of ADP and SH groups. Leone G, Boni P, Vincenti
A (1976). Thromb Haemost 35:249-57. (989191)
- Increased expression of procoagulant activity on the surface of human platelets exposed
to heavy-metal compounds. Goodwin CA, Wheeler-Jones CP, Namiranian S, Bokkala S, Kakkar
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- Thimerosal modulates the agonist-specific cytosolic Ca2+ oscillatory patterns
in single pancreatic acinar cells of mouse. Wu J, Takeo T, Kamimura N, Wada J, Suga S,
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- The effects of thimerosal on the purified InsP3 receptor. Mezna M, Longland CL,
Michelangeli F (1998). Biochem Soc Trans 26:S290. (9766009)
- Fertilisation and thimerosal stimulate similar calcium spiking patterns in mouse oocytes
but by separate mechanisms. Cheek et al., (1993). Development 119:179-189.
(8275854)
- The thiol reagent, thimerosal induces intracellular calcium oscillations in mature human
oocytes. Herbert M, Murdoch AP, Gillespie JI (1995). Hum Reprod 10:2183-6.
(8567870)
- Sperm, inositol trisphosphate, and thimerosal-induced intracellular Ca2+
elevations in rabbit eggs. Fissore RA, Robl JM (1993). Dev Biol 159:122-30.
(8365556)
- The effects of thimerosal, a sulfhydryl reagent, on phasic myometrial contractions.
Phillippe M (1995). Biochem Biophys Res Commun 211:1-6. (7539999)
- Effects of protein kinase C activation and inhibition on sperm-, thimerosal-, and
ryanodine-induced calcium responses of human oocytes. Sousa M, Barros A, Mendoza C,
Tesarik J (1996). Mol Hum Reprod 2:699-708. (9239685)
- Complete activation of porcine oocytes induced by the sulfhydryl reagent, thimerosal.
Machaty Z, Wang WH, Day BN, Prather RS (1997). Biol Reprod 57:1123-7.
(9369179)
- Calcium release and subsequent development induced by modification of sulfhydryl groups
in porcine oocytes. Machaty Z, Wang WH, Day BN, Prather RS (1999). Biol Reprod
60:1384-91. (10330097)
- Time course of cortical and zona reactions of pig oocytes upon intracellular calcium
increase induced by thimerosal. Wang WH, Machaty Z, Abeydeera LR, Prather RS, Day BN
(1999). Zygote 7:79-86. (10216920)
- Intracellular calcium responses in bovine oocytes induced by spermatozoa and by
reagents. Nakada K, Mizuno J (1998). Theriogenology 50:269-82.
(10734495)
D. Allergic
Responses to Thimerosal in Vaccines and Opthalmics
- Thiomersal allergy and vaccination reactions. Cox and Forsyth (1988). Contact
Dermatitis 18:229-233. (3378430)
- Multicenter survey related to the frequency of positive patch tests with mercury and
thiomersal. Lachapelle et al., (1988). Ann. Dermatol. Venereol. 115:793-796.
(2974266)
- Thimerosal: a hidden allergen in opthalmology. Tosti and Tosti (1988). Contact
Dermatitis 18:268-273. (3416589)
- Toxic effects of opthalmic preservatives on cultured rabbit corneal epithelium. Simmons
et al., (1988). Am. J. Optom. Physiol. Opt. 65:867-873. (3252733)
- Cytotoxicity and mutagenicity of opthalmic solution preservatives and UVA radiation in
L5178Y cells. Withrow et al., (1989). Photochem. Photobiol. 50:385-389.
(2780830)
- Reactions to thimerosal in hepatitis B vaccines. Rietschel and Adams (1990). Dermatol.
Clin. 8:161-164. (2137393)
- Patch and prick test study of 593 healthy subjects. Seidenari et al., (1990). Contact
Dermatitis 23:162-167. (2282794)
- A probable role for vaccines containing thimerosal in thimerosal hypersensitivity. Osawa
et al., (1991). Contact Dermatitis 24:178-182. (1868700)
- Cytotoxicity of opthalmic preservatives on human corneal epithelium. Tripathi et al.,
(1992). Lens Eye Toxic Res. 9:361-375. (1301792)
- Ethylmercuric chloride: the responsible agent in thimerosal hypersensitivity. Pirker et
al., (1993). Contact Dermatitis 29:152-154. (8222628)
- Thimerosal induces toxic reaction in non-sensitized animals. Uchida et al., (1994). Int.
Arch. Allergy Immunol. 104:296-301. (7518269)
- Frequency of sensitization to 13 common preservatives in Switzerland. Swiss Contact
Dermititis Research Group. Perrenoud et al., (1994). Contact Dermatitis 30:276-279.
(8088140)
- Sensitization to thimerosal (Merthiolate) is still present today. van 't Veen and van
Joost (1994). Contact Dermatitis 31:293-298. (7867326)
- Sensitization to thimerosal and previous vaccination. Schafer et al., (1995). Contact
Dermatitis 32:114-116. (7758313)
- Hypersensitivity to thimerosal: the sensitizing moiety. Goncalo et al., (1996). Contact
Dermatitis 34:201-203. (8833465)
- Contact allergy in patients with periorbital eczema: an analysis of allergens. Data
recorded by the Information Network of the Departments of Dermatology. Ockenfels et al.,
(1997). Dermatology 195:119-124. (9310716)
- Thimerosal positives: the role of SH groups and divalent ions. Santucci et al., (1998). Contact
Dermatitis 39:123-126. (9771985)
- Contact dermatitis in children: 6 years experience (1992-1997). Romaguera and Vilaplana
(1998). Contact Dermatitis 39:277-280. (9874017)
- Sensitization to thimerosal in atopic children. Patrizi et al., (1999). Contact
Dermatitis 40:94-97. (10048654)