Vaccines And Sudden Infant Death Syndrome
http://vaccineriskawareness.com
"Administration of Diphtheria-Petussis-Tetanus Toxoid (DPT) can cause temporary liver dysfunctions in infants similar to those resulting from viral hepatitis, and inoculation of killed Bordetella pertussis organisms makes some strains of mice 200 times more sensitive to histamine and three to five times as sensitive to endotoxins for approximately 14 days” - Am J. Dis Child 1955; 89:701-716 , http://archpedi.jamanetwork.com/article.aspx?articleid=498098#References
According to a Connaught vaccine manufacturer's data sheet
for old style DPT vaccine,
'Sudden-infant-death-syndrome (SIDS) has been reported following administration
of vaccines containing diphtheria and tetanus toxoids and pertussis vaccine. The
significance of these reports is not clear. It should be borne in mind that the
three primary immunizing doses of these vaccines are usually administered to
infants between the age of 2 and 6 months and that approximately 85% of SIDS
cases occur in the period from 1 through 6 months of age with the peak incidence
at age 2 to 4 months.'
While they admit that SIDS has occured after vaccination with DPT containing
vaccines, they attempt to brush it off by saying that that is because the
majority of SIDS cases happen in the age range when vaccines are being given.
This isn't much of a defence, since it may be because of the shots that the SIDS
occurs in that age group. Evidence suggests this to be the case (I'll discuss
that further along in this article). Even the CDC admit the startling
syncrinicity of the timing between SIDS and vaccines, but then discount it as
'coincidence' and say that studies show the two are not linked, while ignoring
all the studies that say they are.
'From 2 to 4 months old, babies begin their primary course of vaccinations. This
is also the peak age for sudden infant death syndrome (SIDS). The timing of
these two events has led some people to believe they might be related.' -
http://www.cdc.gov/vaccinesafety/Concerns/sids_faq.html
Later manufacturer's data sheets have become more mild in their interpretation
of vaccine side-effects and many fail to mention SIDS, although they do admit to
deaths after vaccination. According to Evans Medical, the Wellcome Foundation,
product no. 0039/0474,
'Neurological events have occasionally been observed following the
administration of pertussis-containing vaccines. The events reported do not
appear to constitute a single, identifiable clinical syndrome but include
isolated febrile convulsions, infantile spasms, episodes of persistent screaming
and severe encephalopathy resulting in permanent brain damage or death. These
events cannot be distinguished from those occurring in unvaccinated children of
similar age. In the absence of a common, identifiable pathological mechanism, it
is not possible to produce reliable estimates of the incidence of neurological
events attributable to pertussis vaccination per Se.'
They state that severe screaming occurs, along with encepholopathy, permanent
brain damage and death, yet attempt to brush that off by claiming unvaccinated
babies have the same symptoms, which is pure nonsense.
1. Authorities have only ever done one double-blind controlled study on
vaccination, in the 1970's, which showed more TB in the vaccine group. This type
of study, which is the only type to get any answers of scientific value, has
never been repeated. Doctors say that to withhold vaccines from children would
be unethical, therefore they never study unvaccinated children.
'Unvaccinated' in their terms means children who have not had DPT containing
vaccines, but have had other vaccines such as polio or Hepatitis B. It is known
that ALL vaccines can cause screaming, encephalitis and death, so that would
make it seem as if all babies are like that and it isn't down to the DPT.
2. Since they never study totally unvaccinated children, they cannot say that
encephalitis and deaths occur in these groups.
3. Since the encephalitis, screaming and deaths occurs AFTER vaccination and not
before, this surely is a clue that the vaccine had something to do with it. If a
person became ill with something prior to any shots being given, one could
obviously not blame vaccination. There would be another cause. But if the
reoccuring factor is, the child has been vaccinated, began screaming, failed to
eat and then died of 'SIDS' (as is the pattern many times over), then it is good
medical practice to at least look at vaccination as a possible cause.
The medical profession and government bodies responsible for investigating SIDS
are run by the Department of Health, and therefore they never look at
vaccination as a possibility, even though the vaccines are being given when SIDS
is peaking in incidence, when manufacturer's admit an association, when there
have been dozens of journal papers suggesting a link and most importantly, when
mothers are screaming
'My baby was vaccinated and he died. I know it wasn't SIDS.'
These mothers are ignored, told they are grieving, 'looking for someone to
blame', or even worse, if the baby showed obvious vaccine injury such as retinal
haemorraging, they are accused of causing the baby's death themselves. In recent
years there has been clusters of parents accused of shaking their own children,
like in the Sally Clark case where she lost one baby 24 hours after his DPT and
another baby on the same day as his vaccinations. She served time for a crime
she didn't commit and although later exonorated, found the trauma too much to
bear and committed suicide.
Her third child, a boy, was not vaccinated during his infancy and he remains
healthy.
Other possible causes are looked at: whether the parents smoke, what position
the baby sleeps in, whether he has a dummy or not, if he's breast fed, whether
he co-sleeps, even what his mattress is made out of. But they're not looking at
the one immunological challenge that nearly all babies go through, vaccination.
Baby Deaths
Charlotte And Vance
Last year a Gloucester couple lost their baby daughter,
Charlotte, a few days after her first vaccinations.
Charlotte, who was born on May 13 last year, had a combined jab for diphtheria,
tetanus, whooping cough and polio, along with Hib meningitis and meningitis C
vaccinations.
Carrie, 22, said: "Viral meningitis showed on Charlotte's autopsy but the final
results came through as cot death.
"We're wondering if the vaccinations caused her death. She had them on Tuesday
and she died the following Monday.
"More babies suffer from cot deaths in their first two to four months and that's
when they have these injections."
Carrie and Dan live with Dan's father, David, who added: "It could have been a
bad batch of vaccinations. We don't know what to believe - but they're not going
to tell us."
Carrie found Charlotte dead in her cot at 7.30am on July 17.
She said: "The day before, Charlotte was fine. She'd drunk all her milk and had
her daily feed before I put her to bed at the usual time - between 9pm and
10pm.
"Dan slept downstairs because Josie was in our bed. I found Charlotte the
following morning. When I woke at 7.30am I thought, 'Hang on, she usually wakes
up at 6am for her feed'.
"I noticed she had some blood coming from her nose. She wasn't moving or
breathing and I screamed for Dan.
"We were trying to resuscitate her and I was screaming down the phone to the
ambulance service asking, 'Where are the paramedics?'
"They took 15 minutes to arrive. We kept doing resuscitation but there was no
sign of life.
"Deep down we knew she'd gone but didn't want to believe it.
"When the paramedics came we thought there could still be a chance, but they did
resuscitation for half an hour and then rang the police."
Carrie denied smoking had affected her baby. She said: "If we had a fag we'd go
into the kitchen and open a window or go outside. So did everyone else.
"We didn't want any of the children breathing in smoke and we didn't smoke in
the car if the girls were there. We even had 'no smoking' signs on the door."
Charlotte wasn't planned but her parents were happy with the pregnancy.
Carrie said: "Everything was all right after the birth. She was a healthy,
bubbly baby."
Dan added: "She was quiet and she was always smiling at you."
At the inquest, pathologist Dr Phillip Cox gave the cause of death as sudden
infant death syndrome.
The inquest heard Charlotte had been born normally after a full-term pregnancy
and had been developing well, according to a medical report into her death.
(Taken from the Gloucestershire Echo, 15 January 2007).
Shelly Walker, from the USA, also lost her 4 month old son less than 3 days
after his vaccinations. 2 other babies who were injected with the same batch of
vaccine also died.
Shelly thinks it's no coincidence that her 4-month-old son died within days of
receiving injections to prevent serious childhood illnesses, including
diphtheria, tetanus, pertussis, hepatitis B, polio, rotavirus and invasive
pneumococcal disease.
"My baby was so healthy," said Shelly Walker, 39, of Hayden. "He was extremely
full of life, energy and vitality."
Nevertheless, early on the morning of Sept. 15, less than three days after Vance
Vernon Walker received a round of vaccines at Lakeside Pediatric and Adolescent
Medicine in Coeur d'Alene, his mother awoke to a nightmare.
"It was about 5:15 a.m. I woke up and thought, 'He's not making any noise!' "
Walker recalled. "I went to pick him up and then I screamed."
Her 16 1/2-pound boy was warm and his lips were still pink, but he wasn't
moving. Blood was crusted beneath his eyes, and his clothes and toys were
covered with a bloody froth.
As her husband, Brian, 46, called 911, Walker worked frantically to resuscitate
their child. But in the emergency room at Kootenai Medical Center, doctors said
Vance had been dead for several hours.
"I grabbed my baby in my arms and held him up and I screamed, 'How in the hell
did this happen?' " Walker said. "Was it the vaccines?"
Medical officials from the CDC and the federal Food and Drug Administration are
working to answer that question for the Walkers and for families of two other
babies who died within six weeks of each other.
Two of the deaths have been logged in the voluntary Vaccine Adverse Event
Reporting System –VAERS – jointly operated by the CDC and FDA, agency officials
said. But Dr. Robert West, the Kootenai County coroner, confirmed that three
infants died this fall within days of immunization.
Parents of the other babies could not be reached for comment.
Autopsies failed to detect any specific vaccine reactions, West said, forcing a
determination of SIDS – a "diagnosis of exclusion," he noted. (Taken from the
spokesmansreview.com).
Two Month Baby Girl Dies
'Our beautiful daughter was born on February 14 and died on
April 17. What was unusual was that earlier on the day she died I had taken her
to the Military Base hospital for her two month checkup. The doctor told me that
she was just perfect. Then the doctor said that she needed four shots. I replied
Four!? She assured me that it was completely normal and that it was better to
give her all at such an early age (because she wouldn't remember the shots).
That evening after feeding [our daughter] we laid her down to sleep and checked
on her 45 minutes later and discovered her dead. I told the police, coroner and
investigators that I thought it was the shots because she was perfectly fine
that day and before the shots. But after 3 weeks we finally got the answer from
the autopsy that it was indeed SIDS. To this day I believe that it was the shots
and no one can convince me otherwise.'
ANON.
Sabra Cline
Sabra has been given her first DPT shot on June 17 1985 in Tulsa, and over the next 48 hours the baby suffered a variety of puzzling symptoms: twitches and seizures, and a coolness to the skin, which also was turning blue
Mrs. Cline said she was aware of the mild symptoms which might be associated with a DPT shot, such as a mild fever and crankiness. But her baby wasn't experiencing those; these were much more frightening.
The infant was taken to a hospital in Muskogee, but Cline feels nothing could have been done by then to save her. "I was holding her when she died," the woman recalls.
At first, it was thought that Sabra had died from Sudden Infant Death Syndrome.
"I said 'No,' my baby's death was associated with the DPT shot. It had to be be," Cline says.
Official autopsy results released seven weeks later listed the cause of death as a swelling and inflammation of the brain tissue.
SIDS IS NOT A DIAGNOSIS!!
You may have noticed in both of those cases, pathologists
determined sudden infant death syndrome to be the cause of death.
SIDS is NOT a diagnosis! It is merely a term to describe a baby who has died
suddenly when they don't know why. It is an UNEXPLAINED infant death. They
cannot say 'SIDS was the cause of death', when they don't know what causes SIDS,
so it may well have been a vaccination. According to the New England Journal of
Medicine:
'In 1969, a National Institutes of Health consensus conference led to the
first standardized definition of sudden infant death as the “sudden death of an
infant or young child, which is unexpected by history, and in which a thorough
post mortem examination fails to demonstrate an adequate cause of death.' (N
Engl J Med. 2009 Aug 20; 361(8): 795–805. ).
Increasingly, more and more pathologists are using the terms SIDS on death
certificates to explain away deaths they can't explain and it shouldn't be done.
Any baby who has died suddenly despite being previously healthy, should be
throughly investigated, including it's vaccination history.
Evidence That The Age Distribution Of SIDS Is Vaccine Related
So, is any of this more than just theory?
Between the years of 1970 and 1974 in Japan, there were 37 infant deaths
directly after vaccination. Because of this, a group of doctors decided to
boycott vaccines and vaccination was stopped altogether for two months. After
that two months, the vaccination starting age was lifted to 2 years old, meaning
that no child recieved a vaccine until after their 2nd birthday. Japan jumped
from 17th place in child mortality to the lowest child mortality in the world.
SIDS had disappeared.
In 1988 the vaccination age was lowered to 3 months old, and the SIDS rate rose
again.
Cherry et al (1988), Pediatrics 81:6 Part 11 (June 1988) Supplement pp 936-984,
said he found this to be 'instructive', suggesting a link between the Japanese
vaccination schedule and SIDS.
Interestingly, Noble et al. (1987) who spent some 2 weeks in Japan studying the
acellular whooping vaccine there, wrote: "It is difficult to exclude pertussis
vaccines as a causal factor even when other etiologies are suggested,
particularly when the adverse events occur in close temporal association with
vaccination".
The same thing has happened in other countries when vaccination is stopped or
delayed.
For instance, in the UK in 1975 and 1976, there was publicity about the whooping
cough vaccine causing brain damage, so the vaccination rate fell to only 30%,
and only 10% in some areas. In line with this, the infant mortality rate also
fell.
By 1977, the controversy was starting to simmer down so vaccination rates began
to climb up. And yes, you've guessed right, the infant mortality rate
increased.
McFarlane (1982) wrote:
"The postneonatal mortality fell markedly in 1976, the year in which a sharp
decline in perinatal mortality rate began. Between 1976 and 1979, however,
neither the late nor the postneonatal mortality rates fell any further. Indeed,
the postneonatal mortality rate increased ,slightly among babies born in 1977.'
This correlates exactly with the rise and fall of vaccination levels.
Fine and Clarkson (1982) wrote "...it is surprising that the interepidemic
period did not decrease after the 1974 fall in vaccine uptake."
They were expecting unvaccinated children to come down with whooping cough in
large numbers. Instead, they had the lowest ever recorded number of whooping
cough hospitalisations and deaths.
More recently in India there has been a drop in infant mortality rates, also in
line with decreasing vaccination:
Two recent health surveys carried out by the Government have thrown up mixed
results. While one reports that the Infant Mortality Rate has fallen below 60
for the first time in the country, the worrying sign is that the already low
immunisation rates are showing further decline.
The most alarming is the case of Uttar Pradesh, which shows a fall in
immunisation from 43.7 per cent in 1998-99 to 28.1 per cent in the latest data.
In 1998-1999, 54 per cent of the children in the country were reported to be
fully immunised. But a district household survey 2002-2004, the data for which
was released last month, shows a decline in this to 47.6 per cent. In 1989-99,
India had one-third of the world?s non-immunised children.
Immunisation rates seem to have fallen across the country, including Uttar
Pradesh and Bihar, which account for 40 per cent of the total children in the
age group of zero to one who need immunisation. But unlike Uttar Pradesh, Bihar
has shown only a marginal decline, from 24.4 to 22.4 per cent.
Experts believe that the focus on polio eradication, at the cost of routine
immunisation, could have contributed to the decline.
The other states showing low figures are Rajasthan (25.4 per cent), Tripura
(26.7 per cent), Jharkhand (29.3 per cent) and Madhya Pradesh (32.5 per cent).
The states at the other end of the spectrum are Tamil Nadu (with an immunisation
rate of 92.1 per cent), Kerala (81.2 per cent), Pondicherry (89.4 per cent), Goa
(81.5 per cent) and Himachal Pradesh (79.4 per cent).
There is good news, however, on the infant mortality front. For the first time,
India has reported IMR below 60, with the survey from Registrar General of India
released recently showing 58 deaths per 1,000 live births in the country.
Though the rates are still high compared to other countries, the figures have
shown decline from 68/1,000 live births in 2000, and 60/1,000 live births in
2004.' (Indian Express).
i.e. Vaccination rates FELL from 54% in 1999 to 47.6% now and the infant
mortality rate has fallen from 68 per 1000 live births in 2000, to 58 per 1000
now.
I'd say that's clear evidence of a link between childhood vaccination and infant
mortality.
Medical Research Into Vaccines and SIDS
A study in Pediatr Infect Dis. 1983 Jan-Feb;2(1):7-11. PMID:
6835859; UI: 83169234, was to determine whether or not DPT caused SIDS. They
contacted the parents of 145 SIDS victims who died in LA between January 1979
and August 1980. Of these, 53 babies had had DPT vaccination. Of those 53, 27
had been given a DPT shot within 28 days of death, 17 deaths occured within 1
week of the vaccination and 6 deaths within 24 hours. This was significantly
more than expected and points to a temporal relationship between DPT and SIDS.
In addition to this there were also 46 babies who died who didn't have DPT but
had visited a clinic before they died (possibly for other vaccines?). 40 of them
died within 28 days of the visit, 22 a week after the visit and seven on the
third day after the visit.
However, the researchers refused to have a control group (i.e. babies who died
of SIDS who had recieved NO vaccines), because they said
'There should be no temporal association between DPT immunization and SIDS.'
So they were never able to get a truly accurate picture, as is the case with all
vaccine 'science'.
Other studies include:
Torch, W.S., 1982. Diphtheria-pertussis-tetanus (DPT) immunization: a potential
cause of the Sudden Infant Death Syndrome (SIDS). Neurology; 32(4): A169
abstract).
Torch, W.C., 1986 a. Characteristics of diphtheria-pertussis-tetanus (DPT)
postvaccinal deaths and DPT-caused Sudden Infant Deaths Syndrome (SIDS): a
review. Neurology (suppl 1); 36: 148 (abstract).
Torch, W.C., 1986 b. Diphtheria-pertussis-tetanus (DPT) imunization may be an
unrecognized cause of Sudden Infant Death (SIDS) and Near-Miss Syndrome (NMS):
12 case reports. Neurology (suppl 1); 36: 149 (abstract).
Cherry, J.D. (1988), Brunell, P.A., Golden, G.S., Karzon, D.T., (1988), Report
of the task force on pertussis and pertussis immunization, Pediatrics 81:6 Part
11 (June 1988) Supplement pp 936-984.
Excerpt: The rate of severe reactions does not differ significantly between the
acellular and whole-cell vaccines when used at 24 months of age. The decrease in
severe reactions is slight, if any. The category "sudden death" is also
instructive in that the entity disappeared following both whole-cell and
acellular vaccines when immunisation was delayed until a child was 24 months of
age. It is clear that delaying the initial vaccination until a child is 24
months, regardless of the type of vaccine, reduces most of the temporally
associated severe adverse events.
Torch, one of the researchers into DPT vaccine and crib death, reported at the
34th annual meeting of the American Academy of Neurology that:
'DPT may be a generally unrecognized major cause of sudden infant and
early childhood death, and that the risks of immunization may outweigh its
potential benefits. A need for re-evaluation and possible modification
of current vaccination procedures is indicated by this study.'
(Torch WC. Diphtheria-pertussis-tetanus (DPT) immunization: a potential cause of
the sudden infant death syndrome (SIDS). American Academy of Neurology, 34th
Annual Meeting, Apr 25-May 1, 1982. Neurology 32(4): pt. 2).
In Current Medicinal Chemistry, although researchers didn't find a causal link,
they did say:
'We selected 110 cases submitted to in-depth histological examination of the
autonomic nervous system and provided with detailed clinical and environmental
information. In 13 cases (11.8%) the death occurred in temporal association with
administration of the hexavalent vaccine (from 1 to 7 days). In none of these
victims congenital developmental alterations of the main nervous structures
regulating the vital functions were observed. Only the hypoplasia of the arcuate
nucleus was present in 5 cases. In one case in particular an acquired hyperacute
encephalitis of the tractus solitarii nucleus was diagnosed in the
brainstem.....we hypothesize that vaccine components could have a direct role in
sparking off a lethal outcome in vulnerable babies.'
Source: Current Medicinal Chemistry, Volume 21, issue 7, DOI 10.2174/09298673113206660289
Sudden Infant Death Following Hexavalent Vaccination: A Neuropathologic Study
Abstract:
We examined a large number of sudden infant death syndrome
victims in order to point out a possible causal relationship between a previous
hexavalent vaccination and the sudden infant death. We selected 110 cases
submitted to in-depth histological examination of the autonomic nervous system
and provided with detailed clinical and environmental information. In 13 cases
(11.8%) the death occurred in temporal association with administration of the
hexavalent vaccine (from 1 to 7 days). In none of these victims congenital
developmental alterations of the main nervous structures regulating the vital
functions were observed. Only the hypoplasia of the arcuate nucleus was present
in 5 cases. In one case in particular an acquired hyperacute encephalitis of the
tractus solitarii nucleus was diagnosed in the brainstem. This study does not
prove a causal relationship between the hexavalent vaccination and SIDS.
However, we hypothesize that vaccine components could have a direct role in
sparking off a lethal outcome in vulnerable babies. In conclusion, we sustain
the need that deaths occurring in a short space of time after hexavalent
vaccination are appropriately investigated and submitted to a post-mortem
examination particularly of the autonomic nervous system by an expert
pathologist to objectively evaluate the possible causative role of the vaccine
in SIDS.
'We hypothesize that vaccine components could have a direct role in sparking
off a lethal outcome in vulnerable babies.' Source: DOI: 10.2174/09298673113206660289
How Would Vaccines Cause SIDS?
All of the ingredients in vaccinations are poisons and
information on them can be found at a poison's unit or environmental health and
safety. When they are contained in other products, we hear how dangerous they
are to our health, for instance, glycol in paint has been attributed to male
infertility, yet this is in vaccines. Mercury was banned from the fillings of
pregnant women because of it's danger, formaldehyde is a known trigger of throat
cancer and everyone knows that aluminium can make you lose your memory.
If you took a single ingredient like this and put it into a baby, I guarantee
you'd have at least one baby who would die as a result. With vaccines, their
tiny little bodies also have to cope with simultaneous viruses and bacteria,
which would never happen in nature, and if you consider that at birth a baby
doesn't really have his own immune system. He is protected by placental
antibodies and his mother's milk. A child's immune system is not fully developed
until the age of 6 years. Just like tiny babies who are at greater risk of dying
of the natural diseases, some babies cannot cope with the vaccine toxins and
just like with any poisoning death, it doesn't always happen instantly. The
body's vital organs can shut down over time.
The Mechanisms of A Vaccine SIDS Death
CYTOKINE STORM
Cytokines are pro-inflammatory messengers that instruct T cells and
macrophages to travel to a site of infection (thus creating inflammation). This
is a mechanism of the immune system to kill off viruses and bacteria and to
repair injury. However, sometimes too many cytokines are formed in a
hyper-immune response and this can cause massive inflammation and tissue damage.
Depending on where the T cells and macrophages are directed to, it can also
cause respiratory failure due to swelling blocking the airway. It can also
cause sepsis and septic shock.
This is called cytokine storm. This has occured after vaccination and infection
as a hyper-immune response.
SIDS cases have increased levels of interleukin-6 in cerebrospinal fluid.
Abstract
Cerebrospinal fluid (CSF) from 20 infants who died
of sudden infant death syndrome (SIDS), 7 cases of infectious death and 5 cases
of violent death were examined with respect to concentrations of interleukin-6
(IL-6). The measurements were performed by ELISA. IL-6 levels in SIDS were
significantly lower than in infectious death (p < 0.02), but significantly
higher than in violent death (p < 0.02). Since IL-6 plays an important role in
immune responses and may induce fever, the findings may suggest that immune
activation plays a role in SIDS. The presence of cytokines in the central
nervous system (CNS) may cause respiratory depression, especially in vulnerable
infants.
Source: Acta
Paediatr. 1995 Feb;84(2):193-6.
Long peptide vaccination can lead to lethality through CD4+ T cell-mediated cytokine storm.
Abstract
The optimization of anticancer therapeutic vaccines can
lead to better efficacy but also to stronger adverse effects. In a mouse model
of antitumor vaccination using a long peptide (LP), which included MHC class I-
and II-restricted male (H-Y) epitopes, we observed unexpected mortality. Mice
with an increased frequency of anti-H-Y CD4 T cells were primed with LP+CpG and
boosted 10 d later. Within hours of boost, they displayed shock-like signs with
high mortality. Serum cytokine levels were high. TNF-alpha secreted by the CD4 T
cells was identified as the key effector molecule. Priming with a short peptide
(SP), which included the MHC class II-restricted epitope, was a more efficient
primer than LP, but did not lead to mortality when used as boost. The high
mortality induced by LP compared with SP was probably related to its specific
ability to be presented by B cells. Finally, targeting the LP sequence to
dendritic cells allowed tumor protection without side effects. Our data: 1)
confirm that the immune system can be very dangerous; 2) caution against the use
of systemic activation of high-frequency Ag-specific T cells as induced by high
doses of LP; and 3) underline the benefit of targeting Ag to dendritic cells.
Source: J
Immunol. 2010 Jul 15;185(2):892-901. doi: 10.4049/jimmunol.1000933. Epub
2010 Jun 11.
Interleukin-2 as a neuromodulator possibly implicated in the physiopathology of sudden infant death syndrome.
Abstract
Dysfunction in vital brainstem centers, including those
controlling cardiorespiratory- and sleep/arousal pathophysiology, is reported
in sudden infant death syndrome (SIDS). Biological mechanisms underlying SIDS,
however, remain unclear. Cytokines are inter-cellular signaling chemicals. They
can interact with neurotransmitters and might thus modify neural and neuroimmune
functions. Cytokines could therefore act as neuromodulators. Interleukin (IL)-2
is a major immune-related cytokine. It has not been previously depicted in vital
brainstem centers. We detected intense neuronal IL-2 immune-reactivity in
the SIDS brainstem, namely in vital neural centers. This IL-2 overexpression
might interfere with neurotransmitters in those critical brainstem centers,
causing disturbed homeostatic control of cardiorespiratory and arousal
responses, possibly leading to SIDS.
Source: Neurosci
Lett. 2010 Aug 16;480(2):122-6. doi: 10.1016/j.neulet.2010.06.021. Epub 2010
Jun 11.
Acute Necrotizing Encephalopathy: An Underrecognized Clinicoradiologic
Disorder
'ANE may also develop secondary to diphtheria, tetanus toxoid, and whole-cell
pertussis (DPTw) vaccination [26]. The DPTw vaccination might result in
increased production of cytokines and cause the alteration of vessel wall
permeability, leading to local breakdown of BBB [38, 44–47].' (BBB is blood
brain barrier).
Source: Mediators of Inflammation
Volume 2015 (2015), Article ID 792578, 10 pages
http://dx.doi.org/10.1155/2015/792578
Possible pathomechanisms of sudden infant death syndrome: key role of chronic hypoxia, infection/inflammation states, cytokine irregularities, and metabolic trauma in genetically predisposed infants.
Abstract
Chronic hypoxia, viral infections/bacterial toxins,
inflammation states, biochemical disorders, and genetic abnormalities are the
most likely trigger of sudden infant death syndrome (SIDS). Autopsy studies have
shown increased pulmonary density of macrophages and markedly more eosinophils
in the lungs accompanied by increased T and B lymphocytes. The elevated levels
of immunoglobulins, about 20% more muscle in the pulmonary arteries, increased
airway smooth muscle cells, and increased fetal hemoglobin and erythropoietin
are evidence of chronic hypoxia before death. Other abnormal findings included
mucosal immune stimulation of the tracheal wall, duodenal mucosa, and palatine
tonsils, and circulating interferon. Low normal or higher blood levels of
cortisol often with petechiae on intrathoracic organs, depleted maternal IgG
antibodies to endotoxin core (EndoCAb) and early IgM EndoCAb triggered, partial
deletions of the C4 gene, and frequent IL-10-592*A polymorphism in SIDS victims
as well as possible hypoxia-induced decreased production of antiinflammatory,
antiimmune, and antifibrotic cytokine IL-10, may be responsible for the
excessive reactions to otherwise harmless infections. In SIDS infants, during
chronic hypoxia and times of infection/inflammation, several proinflammatory
cytokines are released in large quantities, sometimes also representing a
potential source of tissue damage if their production is not sufficiently well
controlled, eg, by pituitary adenylate cyclase-activating polypeptide (PACAP)
and vasoactive intestinal polypeptide (VIP). These proinflammatory cytokines
down-regulate gene expression of major cytochrome P-450 and/or other enzymes
with the specific effects on mRNA levels, protein expression, and enzyme
activity, thus affecting metabolism of several endogenous lipophilic substances,
such as steroids, lipid-soluble vitamins, prostaglandins, leukotrienes,
thromboxanes, and exogenous substances. In SIDS victims, chronic hypoxia, TNF-alpha
and other inflammatory cytokines, and arachidonic acid (AA) as well as n-3
polyunsaturated fatty acids (FA), stimulated and/or augmented superoxide
generation by polymorphonuclear leukocytes, which contributed to tissue damage.
Chronic hypoxia, increased amounts of nonheme iron in the liver and adrenals of
these infants, enhanced activity of CYP2C9 regarded as the functional source of
reactive oxygen species (ROS) in some endothelial cells, and nicotine
accumulation in tissues also intensified production of ROS. These increased
quantities of proinflammatory cytokines, ROS, AA, and nitric oxide (NO) also
resulted in suppression of many CYP450 and other enzymes, eg,
phosphoenolpyruvate carboxykinase (PEPCK), an enzyme important in the metabolism
of FA during gluconeogenesis and glyceroneogenesis. PEPCK deficit found
in SIDS infants (caused also by vitamin A deficiency) and eventually enhanced by
PACAP lipolysis of adipocyte triglycerides resulted in an increased FA level in
blood because of their impaired reesterification to triacylglycerol in
adipocytes. In turn, the overproduction and release of FA into the blood
of SIDS victims could lead to the metabolic syndrome and an early phase of type
2 diabetes. This is probably the reason for the secondary overexpression of the
hepatic CYP2C8/9 content and activity reported in SIDS infants, which
intensified AA metabolism. Pulmonary edema and petechial hemorrhages often
present in SIDS victims may be the result of the vascular leak syndrome caused
by IL-2 and IFN-alpha. Chronic hypoxia with the release of proinflammatory
mediators IL-1alpha, IL-1beta and IL-6, and overloading of the cardiovascular
and respiratory systems due to the narrowing airways and small pulmonary
arteries of these children could also contribute to the development of these
abnormalities. Moreover, chronic hypoxia of SIDS infants induced also production
of hypoxia-inducible factor 1alpha (HIF-1alpha), which stimulated synthesis and
release of different growth factors by vascular endothelial cells and
intensified subclinical inflammatory reactions in the central nervous system,
perhaps potentiated also by PACAP and VIP gene mutations. These processes could
lead to the development of brainstem gliosis and disorders in the release of
neuromediators important for physiologic sleep regulation. All these changes as
well as eventual PACAP abnormalities could result in disturbed homeostatic
control of the cardiovascular and respiratory responses of SIDS victims, which,
combined with the nicotine effects and metabolic trauma, finally lead
to death in these often genetically predisposed children.
Source: Am
J Ther. 2004 Nov-Dec;11(6):517-46.
Urinary tract diseases revealed after DTP vaccination in infants and young children: cytokine irregularities and down-regulation of cytochrome P-450 enzymes induced by the vaccine may uncover latent diseases in genetically predisposed subjects.
Abstract
Prophylactic vaccinations may sometimes shorten the
incubation period of some illnesses and/or convert a latent
infection/inflammation into a clinically apparent disease. Cytokines play a
major role in mediating the inflammatory process in various clinical entities
and represent a potential source of tissue damage if their production is not
sufficiently well controlled. It seems that irregularities in production of
proinflammatory cytokines may be responsible for the abnormalities associated
with full-blown clinical symptoms of various urinary tract diseases observed
after DTP vaccination in 13 infants and young children hospitalized over the
past 24 years. On admission, upper respiratory tract diseases, atopic
dermatitis, and/or latent urinary tract infection/inflammation were found in
these children. It is suggested that the whole-cell pertussis present in DTP
vaccine, acting as an excessive stimulus in these patients, produced symptoms
reminiscent of biologic responses to circulating proinflammatory monokines such
as IL-1beta, TNF-alpha, and IL-6 because earlier it was reported that in vitro
the whole-cell vaccine induced significantly more such cytokine production than
did the acellular pertussis or diphtheria-tetanus-only vaccine. Analysis of the
cellular immune disturbances previously reported in urinary tract
infection/inflammation (increased serum and/or urinary IL-1alpha, IL-1 receptor
antagonist, IL-6 and IL-8), steroid-sensitive nephrotic syndrome (increased
IL-2, IFN-gamma, TNF-alpha, and decreased or increased IL-4, depending on the
cells studied), and atopic dermatitis (decreased IFN-gamma and increased IL-4
production), may suggest that similar subclinical chronic cytokine-mediated
abnormalities produced in the course of latent diseases revealed in our
patients, combined with those caused by DTP vaccination stimulus, were
responsible for the pathomechanism of these clinical entities. This speculation
is in agreement with the reports on the long-lasting induction of cytokine
release and down-regulation of hepatic cytochrome P-450 isoenzyme activities
after administration of DTP vaccine to mice and may be supported by the fact
that TH1 phenotype is associated with the up-regulation of intercellular
adhesion molecule-1 and RANTES, whereas TH2 phenotype is associated with the
up-regulation of the vascular cell adhesion molecule and P-selectin, which are
key players in the migration into inflamed tissues and localization of
lymphocytes and other allergic effector and inflammatory cells. Because several
inflammatory cytokines down-regulate gene expression of major cytochrome P-450
and/or other enzymes with the specific effects on mRNA levels, protein
expression, and enzyme activity, thus affecting the metabolism of several
endogenous lipophilic substances such as steroids, lipid-soluble vitamins,
prostaglandins, leukotrienes, thromboxanes, and exogenous substances, their
irregularities in the body may eventually lead to the flare of latent diseases
in some predisposed subjects. Also, interleukin genetic polymorphisms,
especially the constellation of TNF-alpha and IL-6 genetic variants, might
predispose some infants with infection to a more than usually intense
inflammatory response in the kidneys after vaccination. It seems that the
aforementioned pathomechanism may also be responsible for some cases of sudden
infant death syndrome, which is often preceded by infection/inflammation.
Source:
Am J Ther. 2004 Sep-Oct;11(5):344-53.
Stress
There are several ways in which a baby could die after a
vaccination. They are:
1. Stress. The stress on the body of all the viruses and added toxins, plus the
pain of the injections can cause altered heart rate and breathing patterns. It
is well known that stress can cause heart disease in adults. I firmly believe
that stress can affect babies and children, too. A recent study I read showed
that babies who have a dummy at night, don't seem to die of SIDS. Why, well,
sucking on something comforts them so reduces their adreneline levels and helps
regulate breathing. Of course it would be better if they were suckling from mum,
but these days where most babies are put to bed separately, I can see how a
dummy would reduce stress levels and associated SIDS.
Viera Scheibner is a research scientist who developed the Cotwatch monitor with
her late husband, and she found that baby's breathing patterns were stressed
after vaccination:
'Scientific evidence shows that babies can have severe adverse reactions to
vaccinations at critical intervals following their shots, and that vaccination
is the more likely cause of cot death and shaken baby syndrome.
Dynamics of critical days as part of the dynamics of non-specific stress
syndrome discovered during monitoring with Cotwatch breathing monitor.
Cotwatch was a true breathing monitor, meaning its electronics separated
heartbeat and breathing and only breathing delayed the alarm. The feedback on
breathing from the standard home monitor was from alarms (figure 1), while the
microprocessor-based unit provided computer printouts of the record of breathing
in the form of histograms stacked up at an angle (figure 2) or vertical bars
(figures 3,4), the length of which directly reflected the stress level as
integrals of the weighted apnoea/hypopnoea density (WAHD).
The record of alarms in a baby over a period of five-and-a-half months, from
October 1987 to March 1988 (figure 1), reveals that the stress-induced breathing
pattern did not subside after 21 days following vaccine administration: it was
still continuing on and off (following the critical days) two-and-a-half to
three months later; and before really recovering from the first lot, the child
was given the second injection of DPT and oral polio vaccines. Cotwatch recorded
events in breathing: apnoeas (pauses in breathing) and hypopnoeas (low-volume
breathing, i.e., below 5% of the volume of normal unstressed breathing). The
events were logarithmically weighted (the figures on the vertical axis of the
computer printouts are integrals of the WAHD).
The first two charts in figure 3 are computer printouts of the record of events
in breathing in two babies: baby one, who was given the third DPT (diphtheria-pertussis-tetanus)
vaccine and OPV (oral polio vaccine); and baby two, who was given the first DPT
and OPV. The higher the vertical bar, the higher the stress level in breathing;
figure 3 shows flare-ups of stress-induced breathing day by day from day 0 when
the vaccines were administered and up to the 17th day.
It is obvious that even though baby one reacted much more than baby two, the
flare-ups of stressed breathing followed the same pattern of critical days, the
most important of these being day 2, after which day the stress level went down
and started rising again between days 5 and 7, when the stress level subsided
and started increasing again between days 14-16, subsided again and rose again
between days 19-24, after which it subsided and rose again towards the 28th day
and so on, following closely the pattern of alarms as recorded by a mother of
one baby (figure 1). Days 10 or 11 also emerged as critical days in babies who
reacted strongly, such as baby one. Needless to say, the increased intensity of
reactions after the third DPT injection and OPV reflects the phenomenon of
sensitisation (sensitisation in this context means increased deranged
immunological response or anaphylaxis; and in the case of vaccines it also means
increased susceptibility to the diseases that the vaccines are supposed to
prevent and to a host of unrelated bacterial and viral infections (Parfentjev,
1955; Craighead, 1975; Daum et al., 1989).
The third chart in figure 3 is of 41 actual, randomly listed deaths after DPT
and OPV; it can be seen that the distribution of deaths closely follows the
dynamics of the flare-ups of stressed breathing of babies one and two after the
administration of the DPT vaccine (Bernier et al., 1982, Walker et al., 1987,
Coulter and Fisher, 1991).
Generally speaking, the most fundamental error of judgement displayed by cot
death researchers is that they do not look at what happened to the babies who
succumbed to SIDS, days before they died, and instead they try to identify the
elusive entity of "at risk" babies. The pneumographic studies are done without
any regard to what happens to babies in the first six months and/or one year or
18 months of life when the initial DPT, Hib and polio vaccines and the first MMR
and/or booster vaccines are given.
Vaccinations are mostly ignored in cot death studies. In our experience, the
timing of pneumographic studies is determined by the availability of a bed in
the overnight study unit rather than by looking at what happened to the baby
just before it developed symptoms of stress or started causing alarms on its
monitor.
The notion of false alarms, widely used by those who conduct monitoring of
babies' breathing, has also delayed the understanding of the situation. Alarms
which occurred when the monitored baby did not stop breathing but was breathing
very shallowly are considered false alarms. Leif and I called them "warning
alarms" because they sounded when the monitored babies started having longer and
longer episodes of low-volume breathing, which is the true stress-induced
breathing pattern. A baby who developed pneumonia experienced such "false
alarms" for two weeks before going down with typical symptoms of pneumonia. This
happened about six weeks after the six-month vaccination with DPT and polio
vaccines.
When reactions or deaths occur six weeks after vaccination, they would not be
considered as being caused by vaccination. Yet our records of alarms with
Cotwatch microprocessor computer printouts demonstrate increased stress level
in breathing more than six weeks after vaccination. - Viera Scheibner,
'Vaccination and the dynamics of critical days.'
Immediate Shock
Sometimes a baby can have a shock reaction to a vaccine only minutes or hours after it is given, with difficulty breathing, brain swelling (encephalitis) and blood coming from the eyes or mouth. This happens more frequently in the third world where they often use vaccines which have been banned in Western countries for safety reasons. (See 'Killer Measles Vaccine is Withdrawn' page).
Immune Paralysis
Vaccines challenge the immune system, usually too much, so
in the days after vaccination, when the body is trying to fight the viruses and
heavy metals, it can actually make you more suseptible to the diseases or even
CAUSE the disease.
According to a Hibtiter manufacturer's data sheet from Lederle Laboratories,
1999,
'As with any vaccine, HibTITER may not protect 100% of individuals receiving
the vaccine. Also, as reported with other vaccines, cases of Haemophilus B
disease may occur prior to the onset of the protective effects of the
vaccine.'
And:
'Two children developed Haemophilus B disease after receiving the
two dose primary vaccination schedule. One child became ill at 15 months of
age and the other at 18 months of age....There have been rare reports to the
Vaccine Event Reporting
System (VAERS) of Haemophilus B disease following full primary immunisation.'
In the days or weeks after vaccination, the baby may get HIB disease, blood
poisoning, meningitis, pneumonia and succumb that way, or be left with lots of
respiratory infections as are common with vaccinated children, and then
eventually succumb to SIDS. This is because their immune system has been
weakened and foreign bacteria introduced to the body, so they are unable to stay
healthy.
Dr. Harold Buttram said
'As reported in the New England Journal of Medicine in 1984 [23], 11 healthy
adults had tests involving subpopulations of T-lymphocytes (white blood cells
which mediate immune function) before and after routine tetanus booster
immunizations. The results showed a significant though temporary drop in
T-helper lymphocytes. Special concern rests in the fact that in 4 of the
subjects the T-helper lymphocytes dropped to levels found in active AIDS
patients. If this was the result of a single vaccine in healthy adults, it is
sobering to think of the immune consequences of the multiple vaccines given to
infants with their immature and vulnerable immunity. This study should have
served as a pilot for ongoing vaccine safety studies, but as far as can be
determined from surveys of the medical literature, it has never been repeated.'
Vaccine Induced Scurvy
You've all heard of scurvy, the sailor's disease caused by
malnutrition, well, it may surprise you to learn that vaccines can cause exactly
that.
How?
Dr. Archie Kalokerino's worked as a physician in Australia with aboriginal
people and after a DPT 'immunisation' campaign, he noticed to his horror that
50% - yes HALF - of all babies were dying. After researching what may be the
cause of this huge death rate, he realised that the majority of these babies
were vitamin C deficient and were unable to cope with the vaccine.
Dr. Thomas Levy writes:
'Vaccinations also generally present some degree of toxin insult to the body.
Kalokerinos (1981) observed that vitamin C-deficient Aboriginal infants were
often placed into an acute state of scurvy because of the additional vitamin C
demands placed on their bodies by the vaccination injections, resulting in
sudden death. (Thomas E. Levy, MD, JD, Vitamin C, Infectious Diseases, & Toxins
– Curing The Incurable, 2002).'
After supplementing these babies with vitamin C he found that they no longer
died after vaccination. He was awarded the Nobel Peace Prize for his work.
So how can subclinical scurvy kill?
Vaccination is a major assault to the system and it requires many vitamins to
use in the production of more antibodies (see 'How Your Immune System Works'
page). If there aren't enough vitamins present, then the immune system will
leach them from bones and other vital organs to fight the infection. This can
cause internal bleeding (and subsequent bruising), fractures of the bones and
haemorraging of the retinas and other areas. All of these complications can lead
to death.
It is why some SIDS babies have been found with blood on their mouth or
clothing.
Sometimes the medical profession will accuse the parents of child abuse in an
attempt to explain away the child's injuries, a scenario which is happening more
frequently now that there are more childhood vaccines and more children dying.
According to an article by Jonathan Campbell,
'The "Recommended Daily Allowance" of vitamin C is a small fraction of the
amount we - and infants - really need. Many metabolic processes will be
compromised, but the outward signs won't be obvious. Artery wall repair will
happen more slowly, and the human body compensates for this deficiency with a
sticky plaque called lipoprotein(a) - the root cause of cardiovascular disease.
Antibodies and white blood cells will be built incorrectly or not at all.
Cholesterol, needed for nutrient transport, will not be recycled properly.
For infants, this is deadly. Their little bodies have very little reserves to
draw upon. Without sufficient vitamin C, their immune systems and arteries are
fragile. A single stressful event, a minor fall, a vaccination, a toxic
exposure, or a simple virus or bacterial illness could tip the balance and kill
them. Metabolic failure, heart failure, toxic trauma to vital organs, hemorrhage
- it could happen dozens of ways. Sudden death, with no warning. SIDS.
For vaccinations: In general, I recommend that parents seriously consider not
vaccinating their children, and certainly never to vaccinate against hepatitis
B, because the vaccine has a horrific reputation for harming children, and
hepatitis B is both extremely rare and also quite curable. Vaccines in general
contain both live (but "attenuated") viruses and a brew of toxic chemicals and
preservatives, sometimes including mercury (thimerosal). This places a huge
burden on the child's immune system, and quickly depletes vitamin C. Many SIDS
victims have died shortly after vaccinations.
If you decide to vaccinate your child, increase the vitamin C dose dramatically
several hours before and for several days after the vaccination to prevent
vitamin C depletion and so that the child's immune and detoxification systems
will have a chance to kill the viruses and neutralize the toxins. Demand non-thimerosal,
single-dose, single-virus vaccines; the multiple-virus vaccines such as DPT and
MMP have the worst reputations regarding harm to infants.'
Vaccines and vitamin C deficency are discussed in this medical paper:
Hattersley J, The Answer to Crib Death “Sudden Infant Death Syndrome” (SIDS),
Journal of Orthomolecular Medicine Volume 8, Number 4, 1993, pp.229-245
'Abstract
(1) Two doctors on opposite sides of the globe eliminated crib death among their
patient populations for 40 years using ascorbate supplementation. Unknown to
each other they arrived at the same regimen.
(2) Crib deaths nearly disappeared in Japan in 1975 when first inoculations were
postponed until the 24th month of life.
These findings and their explanation are explored. SID is traced to a
nonspecific or general adaptation stress syndrome defined by Hans Selye. It is
precipitated by a deficiency of ascorbate and also of vitamin B6 and zinc.'
Conclusion
To minimize the risk of SIDS for your baby:
1. Breast feed (this will be discussed on another page).
2. Consider not vaccinating.
3. If you do decide to vaccinate, consider delaying them until your child is 2.
4. If you do decide to vaccinate, supplement your child with vitamin C before
and after the vaccines.
5. If you do decide to vaccinate, make sure the shots don't have thimerosal
(mercury) as an excipient. Space out the jabs and avoid multiple combinations.
6. Even if your child is unvaccinated, if he is sick with anything, was pre-term
or is formula fed, consider giving vitamin C supplementation.
Sudden Unexpected Death in Childhood
Sudden Unexpected Deaths and Vaccinations during the First Two Years of Life in Italy: A Case Series Study
As more and more vaccines have been added to the schedule
and more booster doses given, there are an increasing number of children dying
suddenly when they are over the age of one year. This presented a problem for
the medical community, whose definition of SIDS was a sudden, unexpected death
with no known cause any time during the first year of life. How then could they
explain how previously healthy children over the age of one, just suddenly
dropped dead?
They invented a new term, Sudden Unexpected Death in Childhood, in an attempt to
explain this away.
Ocasionally a child is found to have an underlying medical problem that was not
detected during life, but for some children, research shows that their death is
down to vaccines.
Background
The signal of an association between vaccination in the second year of life with
a hexavalent vaccine and sudden unexpected deaths (SUD) in the two days
following vaccination was reported in Germany in 2003. A study to establish
whether the immunisation with hexavalent vaccines increased the short term risk
of SUD in infants was conducted in Italy.
Methodology/Principal Findings
The reference population comprises around 3 million infants vaccinated in Italy
in the study period 1999–2004 (1.5 million received hexavalent vaccines). Events
of SUD in infants aged 1–23 months were identified through the death
certificates. Vaccination history was retrieved from immunisation registries.
Association between immunisation and death was assessed adopting a case series
design focusing on the risk periods 0–1, 0–7, and 0–14 days after immunisation.
Among the 604 infants who died of SUD, 244 (40%) had received at least one
vaccination. Four deaths occurred within two days from vaccination with the
hexavalent vaccines (RR = 1.5; 95% CI 0.6 to 4.2). The RRs for the risk periods
0–7 and 0–14 were 2.0 (95% CI 1.2 to 3.5) and 1.5 (95% CI 0.9 to 2.4). The
increased risk was limited to the first dose (RR = 2.2; 95% CI 1.1 to 4.4),
whereas no increase was observed for the second and third doses combined.
Source: Traversa G, Spila-Alegiani S, Bianchi C, Ciofi degli Atti M, Frova L, et
al. (2011) Sudden Unexpected Deaths and Vaccinations during the First Two Years
of Life in Italy: A Case Series Study. PLoS ONE 6(1): e16363.
doi:10.1371/journal.pone.0016363
Dr. David Davis MD, Talks about the Link Between Vaccines, PVC and Crib Death
"My suspicion, which is shared by others in my profession, is that the nearly 10,000 SIDS deaths that occur in the US each year are related to one or more of the vaccines that are routinely given to children. The pertussis (whooping cough) vaccine is the
most likely villain , but it could also be one or more of the others. " Dr. R. Mendelsohn - Pediatrician.
Doctor’s Son Dies of SIDS After Vaccines
Nash Brandon Perry was born on 10/26/2015 and taken from us on 1/17/16. Nash was
put down on his back for a nap, when I checked on him about an hour later I
found my son face down, arms straight down at his sides and legs straight out. I
knew something was wrong. I ran, picked him up and immediately saw the horror
that was my precious baby boy dead, blue in the face, with blood coming out of
his nose. I attempted CPR until EMS arrived but it was too late.
Now, maybe it is my grief just looking for answers or something to blame, but there are so many things that just don’t add up. My son never rolled over except twice while throwing a fit and throwing himself around, never while sleeping. I was one room away, he never screamed or cried. Although he was only 2.5 months he had a strong neck. He could hold it up and he could definitely turn it to the side. He had slept the same way many times with no problems. He had no underlying medical problems. My research has found dozens of similar, horrible stories from other parents.
Now, I could just write it off as God’s will or bad luck. But like I said, I’m a scientist, I’m a doctor. There are some very disconcerning similarities between my son’s death and a lot of the SIDS deaths I have read about. A lot of those people were told, “well that’s just coincidence because the highest incidence of SIDS is between 2 and 6 months.” But what if it’s not coincidence? What if we (doctors) are doing routine things to our babies that put them at higher risk that could easily be adjusted to lower the risk, i.e. not giving 8-12 vaccines at one doctors visit to infants. What if Nash, and some other SIDS victims, had some predisposition that made them susceptible to reacting badly to that much vaccine at one time. I am in no way anti-vaccine because they have saved millions of lives, but what if giving them the way we do is contributing to SIDS (our son had his days before). This is just one hypothesis that I want to be able to research. I also want to reach out to other victim’s families, listen to their stories and their hypotheses. During my time as a doctor, I’ve found if you ask the right questions and actually listen to the patient a lot of times they know exactly what is going on or at least have come up with good theories.
What we are asking of you all, those who have shown desire to send us flowers or gifts, instead donate a little money to my new journey. I’m a doctor and now I know my purpose. My purpose is to search for causal relationships between things we are doing that are putting babies at risk of SIDS. To make sure fewer and fewer families have to suffer this horrible tragedy that we are going through . I live in Bexar County which has the highest SIDS rates in all of Texas, so I am confident I am in the right place to gather great data and do good research.
Money donated will be used to fund my research and any money left over will be invested in the companies that are making great innovations /technology to help monitor our babies while they are sleeping and alert us of dangers.
Thank you in advance for your support. I would also like to ask my colleagues
from med school, clinical rotations, residency and current co-workers to please
reach out to me if you want to help with research or any contribution.
In the name of our beautiful Nash, lets Mash Out SIDS!
Source: Before it's News, April 4th 2016.
VAN UK's Comment: Dr Perry was told to stop fundraising for vaccine research and
to take down his social media pages about it.
http://beforeitsnews.com/terrorism/2016/04/doctors-son-dies-of-sids-after-vaccines-2458119.html
http://vaccinecommonsense.com/2016/01/26/804/
Police Officer for 22 Years, Thrown Out of the Force for Whistleblowing about Vaccines and SIDS
I am an ex-Sgt of Police after 20 years in Queensland. Every SIDS mother told me their babies were healthy prior to vaccines and then deteriorated and died after.
Christopher William Savage
POLICE SERGEANT CHRIS SAVAGE BLOWS LID ON VACCINE CRIMES IN AUSTRALIA!!
[vid] POLICE SERGEANT CHRIS SAVAGE BLOWS LID ON VACCINE CRIMES IN AUSTRALIA!!
Mother Explains How Her Daughter Almost Became a SIDS Case when She Stopped Breathing after DTaP and Pneumonia Vaccines - Mother Did CPR to Revive Her
A modified self-controlled case series method to examine association between multidose vaccinations and death.
Abstract
The self-controlled case series method (SCCS) was developed
to analyze the association between a time-varying exposure and an outcome event.
We consider penta- or hexavalent vaccination as the exposure and
unexplained sudden unexpected death (uSUD) as the event. The special situation
of multiple exposures and a terminal event requires adaptation of the standard
SCCS method. This paper proposes a new adaptation, in which observation periods
are truncated according to the vaccination schedule. The new method exploits
known minimum spacings between successive vaccine doses. Its advantage is that
it is very much simpler to apply than the method for censored, perturbed or
curtailed post-event exposures recently introduced. This paper presents a
comparison of these two SCCS methods by simulation studies and an application to
a real data set. In the simulation studies, the age distribution and the
assumed vaccination schedule were based on real data. Only small differences
between the two SCCS methods were observed, although 50 per cent of cases could
not be included in the analysis with the SCCS method with truncated observation
periods. By means of a study including 300 uSUD, a 16-fold risk
increase after the 4th dose could be detected with a power of at least 90 per
cent. A general 2-fold risk increase after vaccination could be detected with a
power of 80 per cent. Reanalysis of data from cases of the German case-control
study on sudden infant death (GeSID) resulted in slightly higher point estimates
using the SCCS methods than the odds ratio obtained by the case-control
analysis.
Source: Stat
Med. 2011 Mar 15;30(6):666-77. doi: 10.1002/sim.4120. Epub 2010 Nov 30.
https://www.ncbi.nlm.nih.gov/pubmed/21337361
Dr Archie Kalokerinos Explains the Sudden Infant Death Syndrome and its Relation to Vaccination
Discussion starts several minutes (10) in as this is part of a much bigger interview, taken in 2004.
Vaccines and SIDS accounts on Camera